The disease works rapidly shutting down the brain. In animals the first symptoms are often behavioral, mooing, lip smacking and licking of the lips, changes in temperament making the animal nervous or aggressive, the disease also causes itching so the animals rub their fur off trying to stop the sensation (“Center for Disease Control and Prevention”). When michael kors womens watches advances symptoms become more clearly representative of the degeneration in the nervous system such as incoordination, difficulty in rising, walking, or standing and loss of body weight despite continued appetite. There are two strains of BSE, the typical strain, which is the strain seen in the majority of cases in the UK epidemic. The typical rose gold michael kors watch is linked to the development of vCJD in humans (Lyman ). The strain can be prevented by eliminating BSE contaminated feed. The second strain is the atypical strain, which is thought to have risen spontaneously because of French research that supports the theory that the atypical BSE represent a sporadic disease.
Variant Creutzfeldt-Jokab Disease destroys brain tissue in the human brain, causing degeneration of the nervous system. The first case of vCJD was reported in the UK in 1996. The disease acts quickly on random parts of the brain causing loss of all nervous function and eventually death. The incubation period in humans is consistent with the first reported infections of BSE in cows in 1984-1986. Bovine to human transmission prolongs the incubation period of the disease so the timetables of the infection of cows and humans match up (“Center for Disease Control and Prevention”). Though there is no way to test for or cure vCJD currently, recent research in 2011, has developed a method of blood testing for the prion. Since the prions lack nucleic acids they do not stimulate an antibody response. It was discovered that prions stick to metal used in surgical tools very well so the experimenters used fine metal particles to stick to the prions in the blood sample. They then used other antibodies to stick to the protein. Scientists developed a human version that could block the disease in mice but is still being tested for safety in humans(“Breakthrough in VCJD blood test”). Though this is a very important step in knowledge of vCJD, it is likely that most of the population was exposed before 1996 and could be carriers who never develop the disease but are still capable of passing it on.